We have a major interest in translational neuroscience and systems neurobiology focusing on disease mechanisms in acute and chronic neuroinflammation. We advocate for diversity and sustainability at various scales and strive for decoding cell type-specific heterogeneity under physiological and pathological conditions in space and time.
In particular, we are wondering why certain cells are more vulnerable than others and what drives cell type reactivity in progressive inflammatory diseases of the muscle and the nervous system. We have a strong interest in supporting repair pathways and helping make cells more resilient in an inflamed tissue environment. Our goal is to maintain homeostasis of cell type integrity and cell-cell communication during lifetime in the context of chronic inflammation.
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